Preliminary experiments suggest conolidine could inhibit distinct ion channels, decreasing neuronal excitability and limiting suffering alerts. This mechanism is especially relevant in neuropathic soreness, where abnormal signaling will cause persistent pain. In addition, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to agony notion. Computational d... https://euripidesm113tya4.mybuzzblog.com/profile